بررسی اولیه تنظیم احساسات و علائم وسواسی جبری
|کد مقاله||سال انتشار||مقاله انگلیسی||ترجمه فارسی||تعداد کلمات|
|34955||2014||6 صفحه PDF||سفارش دهید||5363 کلمه|
Publisher : Elsevier - Science Direct (الزویر - ساینس دایرکت)
Journal : Journal of Obsessive-Compulsive and Related Disorders, Volume 3, Issue 2, April 2014, Pages 109–114
Emotion regulation deficits may contribute to the maintenance of anxiety and distress for individuals with generalized anxiety disorder and other emotional disorders. Recent work suggests a similar model for obsessive-compulsive disorder (OCD); however, the associations between obsessive-compulsive (OC) symptoms and emotion regulation deficits require further empirical support. The present study examined whether understanding of emotion, attention to emotion, negative reactivity (i.e., fear) to emotion, and ability to repair mood states were related to distress associated with OC symptoms. Analyses were conducted using self-report data collected from an undergraduate sample (N=170). Consistent with expectations, OC symptom distress was significantly related to poor understanding of emotions and fear of emotions including both negative (anxiety, anger, and depressed mood) and, unexpectedly, positive emotions. However, excessive attention to emotions and deficits in mood repair were not significantly related to OC symptoms. Comparison of an analog OCD group to controls revealed significantly poorer understanding of emotions and greater fear of emotions in the analog OCD group. Future research on emotion regulation deficits in relation to OC symptoms and OCD appears warranted.
Each particular emotion (e.g., anger, fear, happiness, sadness, disgust, shame) is associated with physiological and behavioral correlates that are typically adaptive in situations that activate that emotion (Panksepp, 1982). Although physiological and behavioral correlates of emotions may occur in a seemingly reflexive way in certain circumstances, emotions are generally subject to elaborative (e.g., cognitive) processing and regulation. Emotion regulation can be defined as the manner in which individuals influence, experience, control, and express their emotions (Gross, 1998). This elaborative processing can modulate the impact of emotions on behavior, fine-tuning responses to a given context; however, poor emotion regulation can also negate the adaptive benefits of emotions. Poor emotion regulation has been associated with worse functioning (Mayer, Salovey, Caruso & Sitarenios, 2001). For example, poor emotion regulation is associated with impaired decision making (Bechara, 2004 and Heilman et al., 2010), deficits in goal-directed behavior (Gray, 2004 and Johnson, 2009), and difficulty reacting adaptively to life׳s demands (i.e., emotional intelligence; Lyons and Schneider, 2005 and Montes-Berges and Augusto, 2007). These impairments can negatively impact social, academic, and career functioning. Mennin, Holaway, Fresco, Moore, and Heimberg (2007) identified three factors involved in emotion dysregulation: (1) poor understanding of emotions, (2) negative reactivity to present emotional state and (3) maladaptive regulation responses. There is evidence that poor emotion regulation may play a role in the development and maintenance of psychopathology (Mennin et al., 2005 and Taylor et al., 1997). Turk, Heimberg, Luterek, Mennin, and Fresco (2005) theorized that poor understanding of emotions diminishes an individuals׳ confidence in their ability to control their emotions. Low confidence spurs fear of their emotional states, prompting the use of maladaptive coping strategies. These maladaptive coping strategies prevent the disconfirmation of negative beliefs about emotion, creating a maladaptive cycle which may lead to or exacerbate psychopathology (Turk et al., 2005). Indeed, emotion regulation deficits have been demonstrated in several psychiatric disorders including: unipolar depression (Rottenberg, Kasch, Gross, & Gotlib, 2002), generalized anxiety disorder (GAD; McLaughlin, Mennin & Farach, 2007; Roemer et al., 2009), eating disorders (Svaldi, Greipenstroh, Tuschen-Caffier & Ehring, 2012), and borderline personality disorder (Glenn & Klonsky, 2009). In a recent meta-analysis of 114 studies, Aldao, Nolen-Hoeksema, and Schweizer (2010) found that internalizing symptoms were correlated with maladaptive emotion regulation strategies (e.g., rumination, avoidance, and suppression). The common presence of emotion regulation deficits across a wide range of disorders suggests that emotion dysregulation may be a transdiagnostic process associated with interference in adaptive function. A number of studies have examined emotion regulation in relation to symptoms of anxiety (Aldao et al., 2010, Mennin et al., 2005, Silk et al., 2003, Suveg and Zeman, 2004, Turk et al., 2005 and Werner et al., 2011) and internalizing disorders (Aldao et al., 2010, Joormann and Gotlib, 2010 and Roemer et al., 2001). Across studies, small to medium correlations have been documented between measures of emotion regulation and worry (Mennin et al., 2005 and Turk et al., 2005) and symptoms of depression (Mennin et al., 2005 and Mennin et al., 2007). Taken together, it appears that poor emotion regulation may be associated with an increase in repetitive efforts to avoid emotional experiences (i.e., worry, rumination, thought suppression). Indeed, poor understanding of emotions, and/or perceptions of emotional experiences as unacceptable may be related to a myriad of maladaptive patterns of behavior aimed at decreasing the presence of strong emotions. Calkins, Berman, and Wilhelm (2013) recently suggested an extension of the emotion regulation model to describe the maintenance of compulsions (repetitive mental or overt acts performed with the goal of reducing anxiety), as seen in obsessive-compulsive disorder (OCD). Specifically, they suggest that individuals with OCD interpret intrusive thoughts as possessing heightened negative salience and potential consequences and that a destructive cycle is created when attempts to quell anxiety in fact raise the amount of attention paid to distressing thoughts and the emotions with which they are associated. They propose that this cycle engenders poor confidence in one׳s ability to regulate distressing moods and thereby leads to the continued use of maladaptive coping strategies (i.e., compulsions). Continued use of compulsions prevents the individual from confronting distressing emotions and inhibits access to discomfirmatory information. This model is consistent with initial data presented by Allen and Barlow (2009) examining the efficacy of exposure to emotion-cues (independent of content) for OCD. Indeed, their intervention sought to reduce avoidance of emotions in individuals with OCD through the use of emotion-provocation exercises. Despite reasons to believe that emotion regulation deficits could play a role in OCD, there is a lack of empirical information on the relation between emotion regulation and OC symptoms. For example, in Aldao et al. (2010) meta-analysis, only 1 of the 114 studies reviewed examined OCD. It is currently unknown if reports of compulsions and/or obsessions (thoughts images or impulses that are experienced as intrusive and distressing, but keep recurring to the individual) are correlated with the experience and regulation of emotions. Therefore, the present study examined the extent to which the three processes hypothesized to characterize emotion dysregulation by Mennin et al. (2007) are related to a broad sample of OC symptoms, including washing, checking, ordering, obsessing, hoarding and mental neutralizing. Specifically, we hypothesized that these OC symptoms would be positively correlated with fear of emotions and negatively correlated with understanding of emotions and the ability to repair distressing mood states. Based on prior findings examining relations between worry and emotion regulation, correlations between these emotion measures and OCD symptoms were expected to be small to medium in magnitude. Further, we hypothesized that, in comparison to controls, participants with heightened OC symptoms would have a poorer understanding of emotions, have a greater fear of emotions, and report less ability to repair distressing mood states.