دانلود مقاله ISI انگلیسی شماره 74983
ترجمه فارسی عنوان مقاله

اثرات مداخله حساسیت اضطراب بر اضطراب، افسردگی و نگرانی: میانجی از طریق تحمل عاطفی

عنوان انگلیسی
The effects of an anxiety sensitivity intervention on anxiety, depression, and worry: Mediation through affect tolerances
کد مقاله سال انتشار تعداد صفحات مقاله انگلیسی
74983 2014 8 صفحه PDF
منبع

Publisher : Elsevier - Science Direct (الزویر - ساینس دایرکت)

Journal : Behaviour Research and Therapy, Volume 59, August 2014, Pages 12–19

ترجمه کلمات کلیدی
حساسیت اضطراب؛ تحمل آشفتگی؛ عدم تحمل ناراحتی - اضطراب؛ افسردگی؛ نگرانی
کلمات کلیدی انگلیسی
Anxiety sensitivity; Distress tolerance; Discomfort intolerance; Anxiety; Depression; Worry
پیش نمایش مقاله
پیش نمایش مقاله  اثرات مداخله حساسیت اضطراب بر اضطراب، افسردگی و نگرانی: میانجی از طریق تحمل عاطفی

چکیده انگلیسی

Recently there has been increased interest in emotional and physical tolerance risk factors for mood and anxiety disorders. Three tolerance risk factors that have been shown to be related are anxiety sensitivity (AS), distress tolerance (DT), and discomfort intolerance (DI). Although previous research has demonstrated these constructs are malleable, no research has investigated the effects of an AS intervention on DT or DI. Further, no studies have investigated whether changes in DT or DI play a role in mood and anxiety symptom amelioration due to an AS intervention. Participants (N = 104), who were selected for elevated levels of AS, completed a single-session computer-assisted AS intervention or a control intervention and follow-up assessments at 1-week and 1-month post intervention. Results revealed that the intervention reduced AS and increased DT, but did not affect DI at the 1-week follow-up. Mediation analyses revealed that changes in AS and DT both mediated changes in symptoms (depression, anxiety, worry) due to the intervention at 1-month follow-up, however, when AS and DT were considered in the same model only the effect via AS remained significant. These results have important implications for the nature of the relationships between AS, DT, and DI as well as the specific mechanistic pathways through which an AS intervention ameliorates symptoms.