دانلود مقاله ISI انگلیسی شماره 62598
ترجمه فارسی عنوان مقاله

اثرات مقاوم به درمان در زبان پریشی سکته مغزی: یک نتیجه از کنترل معنایی ضعیف

عنوان انگلیسی
Refractory effects in stroke aphasia: A consequence of poor semantic control
کد مقاله سال انتشار تعداد صفحات مقاله انگلیسی
62598 2007 15 صفحه PDF
منبع

Publisher : Elsevier - Science Direct (الزویر - ساینس دایرکت)

Journal : Neuropsychologia, Volume 45, Issue 5, 2007, Pages 1065–1079

ترجمه کلمات کلیدی
حافظه معنایی؛ زبان پریشی؛ زوال عقل معنایی؛ اختلال دسترسی؛ مقاوم
کلمات کلیدی انگلیسی
Semantic memory; Aphasia; Semantic dementia; Access impairment; Refractory
پیش نمایش مقاله
پیش نمایش مقاله  اثرات مقاوم به درمان در زبان پریشی سکته مغزی: یک نتیجه از کنترل معنایی ضعیف

چکیده انگلیسی

This study examined the full range of effects associated with “semantic access impairment” – namely, refractory variables (semantic relatedness, speed of presentation and item repetition), inconsistency, the absence of frequency effects and facilitation by cues – in a series of stroke patients with multimodal semantically impairment. By investigating all of these factors in a group of patients who were not specifically selected to show “access” effects, we were able to establish (1) whether this pattern is a common consequence of infarcts that produce semantic impairment and (2) if these symptoms co-occur. All of the patients showed effects of cueing and an absence of frequency effects in comprehension. Patients whose brain damage included the left inferior prefrontal cortex (LIPC) also showed marked effects of refractory variables; in contrast, two patients with temporal–parietal but not frontal lesions were less sensitive to these variables. Parallel results were obtained for cyclical naming and word–picture matching tasks suggesting that the LIPC plays a role in semantic selection as well as lexical retrieval. Rapid presentation and item repetition is likely to have increased the selection demands in both of these tasks in a similar fashion. Unlike patients with classical “semantic access impairment”, our semantically impaired stroke patients showed significant test–retest consistency, indicating that their difficulties did not result from an unpredictable failure of semantic access—instead, their deficits were interpreted as arising from failures of semantic control.