دانلود مقاله ISI انگلیسی شماره 78301
ترجمه فارسی عنوان مقاله

"یادگیری چگونگی برخورد با احساسات متفاوت": داروهای روان گردان به عنوان ابزار اجتماعی شدن در دوران نوجوانی

عنوان انگلیسی
“Learning how to deal with feelings differently”: Psychotropic medications as vehicles of socialization in adolescence
کد مقاله سال انتشار تعداد صفحات مقاله انگلیسی
78301 2015 9 صفحه PDF
منبع

Publisher : Elsevier - Science Direct (الزویر - ساینس دایرکت)

Journal : Social Science & Medicine, Volume 143, October 2015, Pages 311–319

ترجمه کلمات کلیدی
کانادا؛ نوجوانان؛ داروهای روانی؛ سلامت روان گردان؛ مغز نوجوان
کلمات کلیدی انگلیسی
Canada; Adolescence; Psychiatric medications; Mental health; Teenage brain
پیش نمایش مقاله
پیش نمایش مقاله  "یادگیری چگونگی برخورد با احساسات متفاوت": داروهای روان گردان به عنوان ابزار اجتماعی شدن در دوران نوجوانی

چکیده انگلیسی

Drawing from ethnographic research among clinicians working with adolescents at a hospital psychiatric emergency department and outpatient clinic, and with interviews with adolescent psychiatric patients and their parents, we examine how psychiatric medicines function as socializing agents. Although psychiatric medications are thought to exert their main effects through direct biological action on neural circuitry, in fact, their use mobilizes specific kinds of moral discourse and social positioning that may have profound effects on sense of self, personhood, and psychological development. Specifically, our data reveal how clinical discourse around medications aims to enlist adolescents in becoming responsible, emotionally intelligent selves through learning to manage their medications. Among doctors, adolescents and their families, talk about psychiatric medications intertwines narratives of ‘growing up’ and ‘getting well’. Our analysis of case studies from the clinic thus demonstrates that while psychiatric medications are explicitly designed to influence behavior by acting directly on the brain, they also act to structure adolescents' selves and social worlds through indirect, rather than direct causal pathways to the brain.